产品编号 |
bs-47165P-Bio |
英文名称 |
Recombinant human NKG2D protein, N-His-Avi (HEK293), Biotin conjugated
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中文名称 |
生物素标记重组人NKG2D/CD314蛋白 |
别 名 |
NKG2-D type II integral membrane protein; KLRK1; natural killer cell group 2D; NKG2-D isoform b; Klrk1; Nkrp2; NKG2-D-activating NK receptor; NK cell receptor D; Killer cell lectin-like receptor subfamily K; NK lectin-like receptor; CD314; CD 314; CD314 antigen; D12S2489E; Killer cell lectin like receptor subfamily K member 1; Killer cell lectin-like receptor subfamily K member 1; KLR; Klrk1; NKG2 D activating NK receptor; NKG2 D type II integral membrane protein; NKG2-D-activating NK receptor; NKG2D_HUMAN; NKLLR; NKR P2; Nkrp2; Recombinant human NKG2D protein, N-His-Avi (HEK293) / Biotin |
克 隆 号 |
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理论分子量 |
15 kDa |
检测分子量 |
Due to glycosylation, the protein migrates to 36-38 kDa based on Tris-Bis PAGE result. |
浓 度 |
>0.5 mg/ml |
纯 度 |
>95% as determined by Tris-Bis PAGE; >95% as determined by SEC-HPLC |
缓 冲 液 |
Lyophilized from 0.22um filtered solution in PBS (pH7.4) with 5mM DTT. Normally 5% trehalose is added as protectant before Lyophilization. |
保存条件 |
Stored at -70℃ or -20℃. Avoid repeated freeze/thaw cycles. |
注意事项 |
This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications. |
产品介绍 |
This locus represents naturally occurring read-through transcription between the neighboring KLRC4 (killer cell lectin-like receptor subfamily C, member 4) and KLRK1 (killer cell lectin-like receptor subfamily K, member 1) genes on chromosome 12. The read-through transcript includes an alternate 5' exon and lacks a significant portion of the KLRC4 coding sequence, including the start codon, and it thus encodes the KLRK1 protein. [provided by RefSeq, Dec 2010]
NKG2-D出现在免疫细胞上表达的受体,NKG2D属C型凝集素家族跨膜蛋白,广泛表达在NK细胞、CD8+的αβT细胞和γδT细胞表面,可以提高NK细胞对肿瘤细胞的杀伤活性。
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产品图片 |
The purity of Biotinylated Human NKG2D is greater than 95% as determined by SEC-HPLC.
Biotinylated Human NKG2D on Tris-Bis PAGE under reduced condition. The purity is greater than 95%.
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